Now my Dr. wants me to go to an internest and possibly get on meds which I am very opposed to doing. Could my weight loss diet have caused this? Also, I take Evening Primrose oil capsules, pro biotics, fiber DX fiber chews, MSM and Choelius Forskeli. I added Vitamin D3 2000 IU during these last 3 months at the Dr’s suggestion. The only thing I think I may have done differently for my test, I may have taken the Fiber Chew and My eveniing Primrose the night before my test even though I did fast (could they have affected it?)
Unless otherwise stated, statistical analysis was conducted using Prism 6™ software. Values, expressed as means ± SEM, were considered significantly different at p < 0.05. Initial tests were undertaken to ensure that normality and sphericity assumptions were not violated. Subsequently, either one or two way repeated measures ANOVA, or Freidman's test with post-hoc Tukey or Dunnet's correction were performed, to compare changing concentrations of substrates, electrolytes, pH, insulin, breath and urinary βHB: both over time and between study interventions. In Study 2, data from each of the two study visits in each condition (fed and fasted) completed by an individual were included in the analysis.
Personally, I've used Exogenous Ketones to help me through Keto Flu, and to increase my energy levels when doing weight lifting.  As the weights got heavier, I was struggling with shaking while lifting, even though my muscles weren't fatigued.  I started taking drinkable Ketones on heavy lifting days, and the shaking went away and my energy levels increased.
Those hungry fat cells suck up all the available fuel in your blood stream (glucose, fats, ketones). Your body then thinks, “Oh, my god, I am starving. I better eat more and slow my metabolism, so I don’t die.” The problem is, anything you eat gets sucked up into those fat cells around your belly, leading to a vicious cycle of hunger, overeating, fat storage and a slowing down of your metabolism. No wonder we gain weight and can’t lose it.
And the crux of the issue is this: We're continually "fed" the idea that all that's behind the rise in obesity is lack of exercise, or sedentariness. There have certainly been a lot of studies and popular articles suggesting that sitting is our downfall. Instead of effective messages about diet and health that science actually knows to be true, “members of the public are drowned by an unhelpful message about maintaining a ‘healthy weight’ through calorie counting,” the team writes, “and many still wrongly believe that obesity is entirely due to lack of exercise. This false perception is rooted in the Food Industry's Public Relations machinery, which uses tactics chillingly similar to those of big tobacco.”
Table 2 summarizes the dietary composition and nutrient intake of subjects during the study. There was no significant difference in dietary intake between groups at baseline. Subjects in the weight loss group significantly reduced their total energy and fat and significantly increased carbohydrate consumption during the active weight loss period. Energy and nutrient intake did not change in the subjects in the weight maintenance group. That the subjects on the weight loss diet consumed an isocaloric diet from weeks 14 to 16 was supported by the fact that body weight did not vary by >1% during this period. Glycemic load decreased significantly in the weight loss group compared with that in the weight maintenance group, but the glycemic index did not. There was also no change in reported physical activity levels during the study in either the weight loss or weight maintenance groups (data not shown).
Now, Pammet said his “whole mental state” has completely changed. He’s now able to share a side of himself he never knew existed with those around him. “My mindset and thought pattern has changed dramatically everything about the mental side has changed, and I’m very positive, where 11 months ago you wouldn’t even whisper the word positive and associate it with me,” he said.

Still, it is important to recognize that some fruits, like papaya, pineapple, and mango, are higher in natural sugars than other types of fruit. That’s not an issue for most people, but those with type 2 diabetes should be mindful of portion size with these kinds of fruits, due to their potential to spike blood sugar. Fruits like raspberries, apples, and oranges have a relatively lower risk of throwing blood sugar levels out of whack.

As you continue on the diet, you become more adapted or accustomed to making and burning ketones. More ketones are reabsorbed and used for energy, and fewer ketones overflow into the urine.15 After several weeks, as ketone levels rise, evidence suggests your muscles shift to burning fatty acids directly while burning fewer ketones. In this way, ketones are spared for use by other tissues, particularly the brain (which does not burn fatty acids for fuel).16,17
Her clients have had similar success. One woman, for instance, has gone from around 170 pounds to 140 pounds since April without making any initial dietary changes. She’s started to gravitate towards more keto foods over time, but still eats her favorite high-carb treats. As for exercise? Her routine consists of a couple of walks each week, Heverly says.
Do you know people who complain about having a slow metabolism and how they barely eat anything yet still gain weight? Or have you met people who complain about someone they know who can eat whatever he or she wants — including large portions of junk food — due to a fast metabolism and apparently never gain weight. In both cases the individual usually ends by saying, "It's not fair!" These scenarios raise several very good questions: